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Scientists probe the network of processes behind aging

Original: Why do we age — and can science slow it? Aging is not one biological clock. It is a network involving DNA damage, cellul

Aging is being reframed not as a single biological clock but as a network of interacting processes: DNA damage, cellular senescence, mitochondrial dysfunction, chronic inflammation, epigenetic change and declining repair mechanisms. Longevity researchers are now testing which parts of this network can be targeted to slow aging, raising questions about how far interventions might extend healthy lifespan.

Why now: Growing interest in longevity science and whether its findings can translate into therapies that slow aging in humans.

longevity researchcellular senescencemitochondriaepigenetics

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Evidence

• Why do we age — and can science slow it? Aging is not one biological clock. It is a network involving DNA damage, cellular senescence, mitochondrial dysfunction, inflammation, epigenetic change and declining repair. Longevity research is now testing which parts of that network… · nexthorizon · 6

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